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711 2. 5, however its efficiency was further improved. AJR Am J Roent- genol 139:11391144 23. import java. Table 1 Analgesics Drugs Targets Mechanisms Functional consequences Side effects Opioids G-protein coupled μ- δ- κ-receptors cAMP Ca2 currents K currents Excitability of pe- ripheral and central neurons Release of excitatory neurotransmitters μ, δ: sedation, nausea, euphoriare- ward, respiratory depression, constipation κ: dysphoriaaversion, diuresis, sedation NSAIDs Cyclooxygenases (COX-1, COX-2) Prostaglandins Thromboxanes Sensitization of sensory neurons Inhibition of spinal neurons Nonselective: gastrointestinal ulcers, perforation, bleeding, renal impairment COX-2: thrombosis, myocardial infarction, stroke Serotonin agonists G-protein coupled 5-HT receptors 5-HT3: ion channels cAMP (5-HT1) cAMP (5-HT47) PLC (5-HT2) Release of excitatory neuropeptides Neurogenic in- flammation vasoconstriction Myocardial infarction, stroke, peripheral vascular occlusion Antiepileptics Na, Ca2 channels GABA receptors Nacurrents Ca2 currents GABA receptor activity Excitability of pe- ripheral and central neurons Release of excitatory neurotransmitters Sedation, dizziness, cognitive impairment, ataxia, hepatotoxicity, thrombocytopenia Antidepressants Noradrenaline5-HT transporters Na, K channels Noradrenaline 5-HT reuptake Na currents K currents Excitability of pe- ripheral and central neurons Cardiac arrhythmia, myocardial infarction, sedation, nausea, dry mouth, constipation, dizziness, sleep disturbance, blurred vision along all levels of the neuraxis including peripheral and central processes of primary sensory neurons (noci- ceptors), spinal cord (interneurons, projection neurons), brainstem, midbrain, and cortex.

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